Wieckowski MR

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MiPsociety
COST Action CA15203 MitoEAGLE
Evolution-Age-Gender-Lifestyle-Environment: mitochondrial fitness mapping


 

Wieckowski MR


MitoPedia topics: EAGLE 

COST: Member COST WG1: WG1


Name Wieckowski Mariusz R, Prof.
Institution
Mariusz Wieckowski
Laboratory of Bioenergetics and Biomembranes,

Department of Biochemistry,

Nencki Institute of Experimental Biology,

Polish Academy of Sciences, PL

Address 3 Pasteur Str., 02-093
City Warsaw
State/Province
Country Poland
Email m.wieckowski@nencki.gov.pl
Weblink http://duszynskilab.nencki.gov.pl/
O2k-Network Lab PL Warsaw Szewczyk A


Labels: Field of research: Basic 



Publications

 PublishedReference
Flis 2019 Int J Mol Sci2019Flis DJ, Dzik K, Kaczor JJ, Cieminski K, Halon-Golabek M, Antosiewicz J, Wieckowski MR, Ziolkowski W (2019) Swim training modulates mouse skeletal muscle energy metabolism and ameliorates reduction in grip strength in a mouse model of amyotrophic lateral sclerosis. Int J Mol Sci 20:E233.
Gnaiger 2019 MitoFit Preprint Arch2019Gnaiger E, Aasander Frostner E, Abdul Karim N, Abdel-Rahman EA, Abumrad NA, Acuna-Castroviejo D, Adiele RC, et al (2019) Mitochondrial respiratory states and rates. MitoFit Preprint Arch doi:10.26124/mitofit:190001.v6.
Flis 2018 Oxid Med Cell Longev2018Flis DJ, Dzik K, Kaczor JJ, Halon-Golabek M, Antosiewicz J, Wieckowski MR, Ziolkowski W (2018) Swim training modulates skeletal muscle energy metabolism, oxidative stress, and mitochondrial cholesterol content in amyotrophic lateral sclerosis mice. Oxid Med Cell Longev 5940748.
Forkink 2014 Biochim Biophys Acta2014Forkink M, Manjeri GR, Liemburg-Apers DC, Nibbeling E, Blanchard M, Wojtala A, Smeitink JA, Wieckowski MR, Willems PH, Koopman WJ (2014) Mitochondrial hyperpolarization during chronic Complex I inhibition is sustained by low activity of Complex II, III, IV and V. Biochim Biophys Acta 1837:1247-56.
Schoenfeld 2010 Biochim Biophys Acta2010Schönfeld P, Wieckowski MR, Lebiedzinska M, Wojtczak L (2010) Mitochondrial fatty acid oxidation and oxidative stress: Lack of reverse electron transfer-associated production of reactive oxygen species. Biochim Biophys Acta 1797:929-38.

Abstracts

 PublishedReference
Wojtala 2014 Abstract MiP20142014Proteomic analysis of mitochondrial Complex I deficient mouse model - impact of Complex I deficiency on p66Shc-Ser36 phosphorylation pathway in NDUFS4-/- mouse tissues.
Oparka 2014 Abstract MiP20142014Investigation of the mitochondrial functions to understand p66Shc and reactive oxygen species interplay in tumor cells.
Wieckowski 2014 Abstract MiP20142014Understanding the utility and limitations of autopsy samples for mitochondrial studies - evaluation of oxidative stress in the central nervous system of patients with diagnosed or highly probable mitochondrial diseases.
Wieckowski 2013 Abstract MiP20132013Wieckowski MR, Karkucinska-Wieckowska A, Wojtala A, Lebiedzinska M, Pronicki M, Duszynski J (2013) Mitochondrial bioenergetic parameters, reactive oxygen species production and the status of antioxidant defense system can be used to differentiate mitochondrial defects studied in the fibroblasts from patients with various mitochondrial disorders. Mitochondr Physiol Network 18.08.
Wojtala 2013 Abstract MiP20132013Wojtala A, Karkucinska-Wieckowska A, Lebiedzinska M, Duszynski J, Wieckowski MR(2013) Comparative studies of reactive oxygen species production and the level of antioxidant defense system in the fibroblasts derived from patients with defined mitochondrial disorders. Mitochondr Physiol Network 18.08.
Suski 2013 Abstract MiP20132013Suski JM, Karkucinska-Wieckowska A, Pronicki M, Wieckowski MR (2013) Oxidative damage of proteins and the status of antioxidant enzymes in autopsy material from the central nervous system of patients with diagnosed or highly probable mitochondrial diseases. Mitochondr Physiol Network 18.08.
Wojtczak 2012 Abstract Bioblast2012Wojtczak L, Suski JM, Wieckowski MR, Schoenfeld P (2012) A novel putative function of uncoupling proteins. Mitochondr Physiol Network 17.12.

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