Cookies help us deliver our services. By using our services, you agree to our use of cookies. More information

Nakajima T

From Bioblast
Revision as of 09:56, 14 February 2018 by Beno Marija (talk | contribs) (→‎Participated at)
(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Name Nakajima Takayuki,
Institution Hokkaido University

Department of Cardiovascular Medicine, Hokkaido University Graduate School of Medicine

Address 5 Chome Kita 8 Jōnishi, 060-0808
City Sapporo
State/Province
Country Japan
Email [email protected]
Weblink
O2k-Network Lab JP Sapporo Yokota T


Labels:



Publications

 PublishedReference
Aya 2022 Eur J Pharmacol2022Shiraki A, Oyama J, Shimizu T, Nakajima T, Yokota T, Node K (2022) Empagliflozin improves cardiac mitochondrial function and survival through energy regulation in a murine model of heart failure. Eur J Pharmacol 931: 175194.
Nambu 2021 Cardiovasc Res2021Nambu H, Takada S, Maekawa S, Matsumoto J, Kakutani N, Furihata T, Shirakawa R, Katayama T, Nakajima T, Yamanashi K, Obata Y, Nakano I, Tsuda M, Saito A, Fukushima A, Yokota T, Nio-Kobayashi J, Yasui H, Higashikawa K, Kuge Y, Anzai T, Sabe H, Kinugawa S (2021) Inhibition of xanthine oxidase in the acute phase of myocardial infarction prevents skeletal muscle abnormalities and exercise intolerance. Cardiovasc Res 117:805-19.
Shirakawa 2019 Sci Rep2019Shirakawa R, Yokota T, Nakajima T, Takada S, Yamane M, Furihata T, Maekawa S, Nambu H, Katayama T, Fukushima A, Saito A, Ishimori N, Dela F, Kinugawa S, Anzai T (2019) Mitochondrial reactive oxygen species generation in blood cells is associated with disease severity and exercise intolerance in heart failure patients. Sci Rep 9:14709.
Nakajima 2019 Sci Rep2019Nakajima T, Yokota T, Shingu Y, Yamada A, Iba Y, Ujihira K, Wakasa S, Ooka T, Takada S, Shirakawa R, Katayama T, Furihata T, Fukushima A, Matsuoka R, Nishihara H, Dela F, Nakanishi K, Matsui Y, Kinugawa S (2019) Impaired mitochondrial oxidative phosphorylation capacity in epicardial adipose tissue is associated with decreased concentration of adiponectin and severity of coronary atherosclerosis. Sci Rep 9:3535.

Abstracts

 PublishedReference
Nakajima 2017 Europ Heart J2017Mitochondrial dysfunction in epicardial adipose tissue; possible role in progression of coronary artery disease.

Participated at