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Difference between revisions of "Krako Jakovljevic 2021 MitoFit PD"

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{{Publication
{{Publication
|title=Krako Jakovljevic N, Ebanks B, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease. MitoFit Preprints 2021.4. [[doi:10.26124/mitofit:2021-0004]]
|title=Krako Jakovljevic N, Ebanks B, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease. MitoFit Preprints 2021.4. [[doi:10.26124/mitofit:2021-0004]] <!-- Published in [[Krako Jakovljevic_202#_##### |Ā»BEC 2021.#Ā«]] -->
|info=[[File:MitoFit Preprints pdf.png|left|160px|link=https://wiki.oroboros.at/images/1/15/Krako_Jakovljevic_2021_MitoFit_PD.pdf |MitoFit pdf]] [https://wiki.oroboros.at/images/1/15/Krako_Jakovljevic_2021_MitoFit_PD.pdf Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease]
|info=[[File:MitoFit Preprints pdf.png|left|160px|link=https://wiki.oroboros.at/images/1/15/Krako_Jakovljevic_2021_MitoFit_PD.pdf |MitoFit pdf]] [https://wiki.oroboros.at/images/1/15/Krako_Jakovljevic_2021_MitoFit_PD.pdf Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease]
|authors=Krako Jakovljevic Nina, Ebanks Brad, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta
|authors=Krako Jakovljevic Nina, Ebanks Brad, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta
|year=2021-03-03
|year=2021-03-03
|journal=MitoFit Prep
|journal=MitoFit Prep
|abstract=Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinsonā€™s disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression.
|abstract=
<!-- <big>'''Published 2021-##-## in [[Krako Jakovljevic_BEC_2021.# |Ā»BEC 2021.#Ā«]] '''</big>
::: Version 1 ('''v1''') '''2021-03-03''' [File:MitoFit Preprints pdf.png|left|160px|link=https://wiki.oroboros.at/images/1/15/Krako_Jakovljevic_2021_MitoFit_PD.pdf doi:10.26124/mitofit:2021-0004] -->
Ā 
Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinsonā€™s disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression.
|keywords=Parkinsonā€™s Disease, cellular models, mitochondrial homeostasis
|keywords=Parkinsonā€™s Disease, cellular models, mitochondrial homeostasis
|editor=Cardoso LHD
|editor=Cardoso LHD

Revision as of 09:26, 13 September 2021


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Krako Jakovljevic 2021 MitoFit PD

Publications in the MiPMap
Krako Jakovljevic N, Ebanks B, Chakrabarti L, Markovic I, Moisoi N (2021) Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease. MitoFit Preprints 2021.4. doi:10.26124/mitofit:2021-0004

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MitoFit pdf

Mitochondrial homeostasis in cellular models of Parkinsonā€™s Disease

Krako Jakovljevic Nina, Ebanks Brad, Chakrabarti Lisa, Markovic Ivanka, Moisoi Nicoleta (2021-03-03) MitoFit Prep

Abstract: Mitochondrial function is known to be an important factor in maintaining cellular homeostasis and its dysregulation has become a hallmark for multiple disease conditions. This review aims to synthesise the extent of this knowledge by analysing changes of mitochondrial physiology parameters in Parkinsonā€™s disease (PD) and to evaluate the contribution of cellular models of PD in the field. The analysis provided here constitutes a platform for further elucidation of mitochondrial function parameters relative to factors that may potentiate disease progression. ā€¢ Keywords: Parkinsonā€™s Disease, cellular models, mitochondrial homeostasis ā€¢ Bioblast editor: Cardoso LHD ā€¢ O2k-Network Lab: RS Belgrade Lalic NM, UK Nottingham Chakrabarti L, UK Leicester Moisoi N

ORCID: ORCID.png Krako Jakovljevic Nina, ORCID.png Ebanks Brad, ORCID.png Chakrabarti Lisa ORCID.png Markovic Ivanka ORCID.png Moisoi Nicoleta

Labels: Pathology: Parkinson's 

Organism: Human, Mouse, Drosophila