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Difference between revisions of "Clementi Emilio"

From Bioblast
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|firstname=Emilio
|firstname=Emilio
|title=Prof. , Mus. M., M.D., Ph.D.
|title=Prof. , Mus. M., M.D., Ph.D.
|institution=Head, Unit of Clinical Pharmacology,
|institution=Unit of Clinical Pharmacology,
Fatebenefratelli-Sacco Hospital, University  of Milano


Editor in Chief, Pharmacological Research
The European Association for Clinical Pharmacology and Therapeutics, EACPT,
Consulting Editor, Clinical Therapeutics
Member in the Executive Committee,


The European Association for Clinical Pharmacology and Therapeutics, EACPT
Department of Biomedical and Clinical Sciences “L. Sacco”,  


Department of Biomedical and Clinical Sciences “L. Sacco”, University of Milano,
Fatebenefratelli-Sacco Hospital,  


DIBIT-H San Raffaele Institute
DIBIT-H San Raffaele Institute,
 
University of Milano, IT
|address=Via GB Grassi 74
|address=Via GB Grassi 74
|area code=20157
|area code=20157

Revision as of 12:58, 10 December 2018

Name Clementi Emilio, Prof. , Mus. M., M.D., Ph.D.
Institution Unit of Clinical Pharmacology,

The European Association for Clinical Pharmacology and Therapeutics, EACPT,

Department of Biomedical and Clinical Sciences “L. Sacco”,

Fatebenefratelli-Sacco Hospital,

DIBIT-H San Raffaele Institute,

University of Milano, IT

Address Via GB Grassi 74, 20157
City Milan
State/Province
Country Italy
Email [email protected]
Weblink
O2k-Network Lab IT Milan Clementi E


Labels:



Publications

 PublishedReference
Giovarelli 2023 Mol Med2023Giovarelli M, Serati A, Zecchini S, Guelfi F, Clementi E, Mandò C (2023) Cryopreserved placental biopsies maintain mitochondrial activity for high-resolution respirometry. https://doi.org/10.1186/s10020-023-00645-2
Giovarelli 2021 Pharmacol Res2021Giovarelli M, Zecchini S, Catarinella G, Moscheni C, Sartori P, Barbieri C, Roux-Biejat P, Napoli A, Vantaggiato C, Cervia D, Perrotta C, Clementi E, Latella L, De Palma C (2021) Givinostat as metabolic enhancer reverting mitochondrial biogenesis deficit in Duchenne Muscular Dystrophy. Pharmacol Res 170:105751.
BEC 2020.1 doi10.26124bec2020-0001.v12020Gnaiger E et al ― MitoEAGLE Task Group (2020) Mitochondrial physiology. Bioenerg Commun 2020.1. https://doi.org/10.26124/bec:2020-0001.v1
Pedrotti 2019 Sci Adv2019Pedrotti S, Caccia R, Neguembor MV, Garcia-Manteiga JM, Ferri G, de Palma C, Canu T, Giovarelli M, Marra P, Fiocchi A, Molineris I, Raso M, Sanvito F, Doglioni C, Esposito A, Clementi E, Gabellini D (2019) The Suv420h histone methyltransferases regulate PPAR-γ and energy expenditure in response to environmental stimuli. Sci Adv 5:eaav1472.
Pambianco 2016 Cell Rep2016Pambianco S, Giovarelli M, Perrotta C, Zecchini S, Cervia D3, Di Renzo I, Moscheni C, Ripolone M, Violano R, Moggio M, Bassi MT, Puri PL, Latella L, Clementi E, De Palma C (2016) Reversal of defective mitochondrial biogenesis in limb-girdle muscular dystrophy 2D by independent modulation of histone and PGC-1α acetylation. Cell Rep 17:3010-23.
De Palma 2014 Skelet Muscle2014De Palma C, Morisi F, Pambianco S, Assi E, Touvier T, Russo S, Perrotta C, Romanello V, Carnio S, Cappello V, Pellegrino P, Moscheni C, Bassi MT, Sandri M, Cervia D, Clementi E (2014) Deficient nitric oxide signalling impairs skeletal muscle growth and performance: involvement of mitochondrial dysregulation. Skelet Muscle 4:22.
Mandò 2014 Am J Physiol Endocrinol Metab2014Mandò C, De Palma C, Stampalija T, Anelli GM, Figus M, Novielli C, Parisi F, Clementi E, Ferrazzi E, Cetin I (2014) Placental mitochondrial content and function in intrauterine growth restriction and preeclampsia. Am J Physiol Endocrinol Metab 306:E404-13.
De Palma 2010 Cell Death Differ2010De Palma C, Falcone S, Pisoni S, Cipolat S, Panzeri C, Pambianco S, Pisconti A, Allevi R, Bassi MT, Cossu G, Pozzan T, Moncada S, Scorrano L, Brunelli S, Clementi E (2010) Nitric oxide inhibition of Drp1-mediated mitochondrial fission is critical for myogenic differentiation. Cell Death Differ 17:1684-96.
Nisoli 2007 Circ Res2007Nisoli E, Clementi E, Carruba MO, Moncada S (2007) Defective mitochondrial biogenesis: a hallmark of the high cardiovascular risk in the metabolic syndrome? Circ Res 100:795-806.

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