Difference between revisions of "Aon 2015 Antioxid Redox Signal"
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{{Publication | {{Publication | ||
|title=Aon MA, Foster DB (2015) Diabetic cardiomyopathy and the role of mitochondrial dysfunction: novel insights, mechanisms, and therapeutic strategies. Antioxid Redox Signal 22:1499-501. | |title=Aon MA, Foster DB (2015) Diabetic cardiomyopathy and the role of mitochondrial dysfunction: novel insights, mechanisms, and therapeutic strategies. Antioxid Redox Signal 22:1499-501. | ||
|info=[http://www.ncbi.nlm.nih.gov/pubmed/25843129 PMID:25843129] | |info=[http://www.ncbi.nlm.nih.gov/pubmed/25843129 PMID:25843129] | ||
|authors=Aon MA, Foster DB | |authors=Aon MA, Foster DB | ||
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|injuries=Oxidative stress;RONS | |injuries=Oxidative stress;RONS | ||
|diseases=Cardiovascular, Diabetes | |diseases=Cardiovascular, Diabetes | ||
}} | |||
Latest revision as of 11:40, 21 March 2016
| Aon MA, Foster DB (2015) Diabetic cardiomyopathy and the role of mitochondrial dysfunction: novel insights, mechanisms, and therapeutic strategies. Antioxid Redox Signal 22:1499-501. |
Aon MA, Foster DB (2015) Antioxid Redox Signal
Abstract: This Forum addresses the role of mitochondrial dysfunction in the multifactorial nature of diabetic cardiomyopathy (DCM) from multiple angles. Contributors deliver a diverse and in-depth view of the state-of-the-art in DCM, from bench to bedside. What emerges is a picture of mitochondrial dysfunction as a central upstream defect, inflicted on the heart by diabetes. Collectively, the authors pinpoint high-value knowledge gaps, propose new conceptual frameworks, and highlight understudied, but promising, research themes.
Labels: MiParea: mt-Structure;fission;fusion, mtDNA;mt-genetics, Exercise physiology;nutrition;life style, mt-Medicine, Patients, mt-Awareness
Pathology: Cardiovascular, Diabetes
Stress:Oxidative stress;RONS
Organism: Human
