Tretter 2016 Biochim Biophys Acta: Difference between revisions
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{{Publication | {{Publication | ||
|title=Tretter L, Patocs A, Chinopoulos C (2016) Succinate, an intermediate in metabolism, signal transduction, ROS, hypoxia, and tumorigenesis. Biochim Biophys Acta 1857:1086-101. | |title=Tretter L, Patocs A, Chinopoulos C (2016) Succinate, an intermediate in metabolism, signal transduction, ROS, hypoxia, and tumorigenesis. https://doi.org/10.1016/j.bbabio.2016.03.012 | ||
|info=Biochim Biophys Acta 1857:1086-101. [https://www.ncbi.nlm.nih.gov/pubmed/26971832 PMID:26971832 Open Access] | |||
|authors=Tretter L, Patocs A, Chinopoulos C | |authors=Tretter L, Patocs A, Chinopoulos C | ||
|year=2016 | |year=2016 | ||
|journal=Biochim Biophys Acta | |journal=Biochim Biophys Acta | ||
|abstract=Succinate is an important metabolite at the cross-road of several metabolic pathways, also involved in the formation and elimination of reactive oxygen species. However, it is becoming increasingly apparent that its realm extends to epigenetics, tumorigenesis, signal transduction, endo- and paracrine modulation and inflammation. Here we review the pathways encompassing succinate as a metabolite or a signal and how these may interact in normal and pathological conditions. | |abstract=Succinate is an important metabolite at the cross-road of several metabolic pathways, also involved in the formation and elimination of reactive oxygen species. However, it is becoming increasingly apparent that its realm extends to epigenetics, tumorigenesis, signal transduction, endo- and paracrine modulation and inflammation. Here we review the pathways encompassing succinate as a metabolite or a signal and how these may interact in normal and pathological conditions. | ||
|mipnetlab=HU Budapest Chinopoulos C, HU Budapest Tretter L | |||
}} | }} | ||
{{Labeling | {{Labeling | ||
|area=Respiration | |area=Respiration | ||
|pathways=S | |diseases=Cancer | ||
|injuries=Oxidative stress;RONS | |||
|topics=ADP, ATP | |||
|couplingstates=LEAK, OXPHOS | |||
|pathways=N, S, NS | |||
}} | }} | ||
Latest revision as of 11:24, 25 March 2023
| Tretter L, Patocs A, Chinopoulos C (2016) Succinate, an intermediate in metabolism, signal transduction, ROS, hypoxia, and tumorigenesis. https://doi.org/10.1016/j.bbabio.2016.03.012 |
» Biochim Biophys Acta 1857:1086-101. PMID:26971832 Open Access
Tretter L, Patocs A, Chinopoulos C (2016) Biochim Biophys Acta
Abstract: Succinate is an important metabolite at the cross-road of several metabolic pathways, also involved in the formation and elimination of reactive oxygen species. However, it is becoming increasingly apparent that its realm extends to epigenetics, tumorigenesis, signal transduction, endo- and paracrine modulation and inflammation. Here we review the pathways encompassing succinate as a metabolite or a signal and how these may interact in normal and pathological conditions.
• O2k-Network Lab: HU Budapest Chinopoulos C, HU Budapest Tretter L
Labels: MiParea: Respiration
Pathology: Cancer
Stress:Oxidative stress;RONS
Regulation: ADP, ATP
Coupling state: LEAK, OXPHOS
Pathway: N, S, NS
